It was aimed at observing the histochemical basis of physiological experiment about the effectof electroacupuncture(EA)on acute myocardial ischemia(AMI)in this paper.The capillaries ofmyocardium were reflected with both alkaline phosphatase(ALP)and Mg

(2+)

-ATP_(asc).Thirty minafter ligating the left ventricular branch(LVB)

the capillaries stained by ALP were much de-creased in the ischemic group without EA

while in EA group they were increased distinctly.Thenumber and the total length of the capillaries in a 220×320μm ischemic area of the posterior wall(section 10μm)were measured and compared.In ischemic group the number was 17.2±1.65

the length was 634.62±666.24μm

in EA group 22.5+1.44

1187.57+103.69μm

respectively.Both counts were significant differences(P

<

0.05

P

<

0.001).It reflected that EA could improvethe microcirculation and metabolic function of AMI.Mg(2+)-ATP_(asc).Thirty minafter ligating the left ventricular branch(LVB)

the capillaries stained by ALP were much de-creased in the ischemic group without EA

while in EA group they were increased distinctly.Thenumber and the total length of the capillaries in a 220×320μm ischemic area of the posterior wall(section 10μm)were measured and compared.In ischemic group the number was 17.2±1.65

the length was 634.62±666.24μm

in EA group 22.5+1.44

1187.57+103.69μm

respectively.Both counts were significant differences(P

<

0.05

P

<

0.001).It reflected that EA could improvethe microcirculation and metabolic function of AMI.Mg

(2+)

-ATPase was shown on the plasmmembrance of endothelial cell of microvasculature and analysed quantitatively with a Univar scanning microspectrophotometer.After occluding LVB for 30 min its optic density of AMI was de-creased to 106.834-14.06

while it was increased to 210.83+24.88 in EA group

P

<

0.05.Theactivity of Mg(2+)-ATPase was shown on the plasmmembrance of endothelial cell of microvasculature and analysed quantitatively with a Univar scanning microspectrophotometer.After occluding LVB for 30 min its optic density of AMI was de-creased to 106.834-14.06

while it was increased to 210.83+24.88 in EA group

P

<

0.05.Theactivity of Mg

(2+)

-ATPase could increase in transporting Na(?)

k(?)ions through the membrance ofthe endothelial cells of ischemic myocardium.The result was consistent with the physiolgical ex-periment

because EA could regulate the changes of mean repolarization rate induced by AMI andadjust the ion concentration of transmembrane.The catecholamine(CA)in myocardium was lo-cated at sympathetic adrenergic terminals with varicosities containing norepinephrine(NE).Thirty rain after ligating LVB

the fluorescence of adrenergic terminals was decreased and/or de-pleted

but in EA group

the fluorescent varicosities were increased gradually.It was benficial tostrengthing the contractibility of myocardium.