摘要:本研究采用单克隆抗体技术对51例恶性肿瘤患者外周血T 细胞及其亚群进行了针刺前后的观察。实验设计了三个组:恶性肿瘤患者的针刺组和对照组,及正常人对照组。以 OKT_3、OKT_4,OKT_8及OKT_4/OKT_8为观察指标。结果表明,针刺治疗能够提高恶性肿瘤患者外周血 T 细胞及其亚群的百分率,尤以 OKT_4+细胞受针刺影响最明显。Effect of acupuncture on the T-lymphocyte and its subsets from the peripheral blood of pa-tients with malignant neoplasm has been researched in this study.51 patients were devided intotwo groups:one in acupuncture treatment and the other without any treatment.48 healthy adultswere also studied as normal control group.The results showed that the percentages of OKT_3+细胞受针刺影响最明显。
摘要:本文以猫脊髓处的检验刺激所诱发的腓肠神经逆向 C 波大小作为判断初级传入 C 纤维突触前末梢兴奋性的指标。电针“环跳”及“阳陵泉”穴引起该逆向 C 波明显增大。表明初级传入 C 纤维突触前末梢发生了去极化。而荷包牡丹碱、纳络酮及 P 物质抗血清能明显地抑制此针刺效应。提示 GABA,内阿片肽及 P 物质均参与针刺突触前抑制的调节。
摘要:许多研究表明,脑内 P 物质参与针刺镇痛过程;大脑皮层体感Ⅱ区(SⅡ)参与对丘脑中央中核伤害性反应和针刺镇痛效应的下行调节。为了探讨 P 物质是否参与大脑皮层SⅡ区对丘脑的下行调节,本实验观察了电刺激猫 SⅡ区和电针穴位对丘脑中央中核灌流液中 P 物质含量的影响。实验用成年猫,体重1.6~4.1公斤,雌雄不拘。动物随机分为三组:对照组、电针组和 SⅡ区电刺激组,每组分别为10、15和12只。用戊巴比妥钠(40毫克/公斤)麻醉,行开颅术。暴露It was investigated that the effects of electrical stimulation of the somatosensory area Ⅱ(SII)of the cerebral cortex and electroacupuncture on substance P(SP)concentration in the per-fusate from the nucleus centrum medianum(CM)of the thalamus,in order to probe into if SP participates in the SII descending modulation of the nucleus CM and this mechanism of acupunc-ture analgesia.Adult cats were randomly divided into three groups:control,electroacupuncture and electri-cal stimulation of SII.Push-pull perfusion technique was used to collect the perfusate from thenucleus CM.SP contents in the perfusate were determined by radioimmunoassay.The results showed that SP contents in the perfusate have not statistically significantchanges before and after the electrical stimulation of SII or electroacupuncture,suggesting thatthe SP release from the nucleus CM was not influenced by the stimulation of SII or elec-troacupuncture.It is indicated that SP may not associate with the SII descending modulation ofthe nucleus CM and this mechanism of acupuncture analgesia.
摘要:以碱性磷酸酶(ALP)、镁激活的腺苷三磷酸酶(Mg(2+)-ALP_(ase))和儿茶酚胺(CA)荧光组化方法观察了电针对急性心肌缺血作用的机理。结扎左冠状动脉心室支30分钟后,缺血心肌中以 ATP 显示的毛细血管大为减少,长度缩短;而缺血电针组则有显著意义增加,说明电针对缺血心肌微循环和转运功能的改善。Mg(2+)-ALP_(ase))和儿茶酚胺(CA)荧光组化方法观察了电针对急性心肌缺血作用的机理。结扎左冠状动脉心室支30分钟后,缺血心肌中以 ATP 显示的毛细血管大为减少,长度缩短;而缺血电针组则有显著意义增加,说明电针对缺血心肌微循环和转运功能的改善。Mg(2+)-ATP_(ase)可显示心肌微血管的质膜和基膜;经显微分光光度计测量,Mg(2+)-ATP_(ase)可显示心肌微血管的质膜和基膜;经显微分光光度计测量,Mg(2+)-ATP_(ase)在缺血电针组比缺血对照组光密度增加,这可加强缺血心肌 Na(2+)-ATP_(ase)在缺血电针组比缺血对照组光密度增加,这可加强缺血心肌 Na+K+K+离子跨膜转动功能。心肌儿茶酚胺荧光所显示的交感肾上腺素能膨体神经末梢;缺血后减少或耗竭,电针组则较多而亮,这有益于加强缺血心肌的收缩力。It was aimed at observing the histochemical basis of physiological experiment about the effectof electroacupuncture(EA)on acute myocardial ischemia(AMI)in this paper.The capillaries ofmyocardium were reflected with both alkaline phosphatase(ALP)and Mg+离子跨膜转动功能。心肌儿茶酚胺荧光所显示的交感肾上腺素能膨体神经末梢;缺血后减少或耗竭,电针组则较多而亮,这有益于加强缺血心肌的收缩力。
摘要:以乌头碱造成家兔实验性心律失常模型,针刺“内关”穴观察对心律失常恢复时间的影响。在肯定针刺“内关”疗效的基础上用 EDTA 溶液络合掉心包经曲泽穴处的 Ca(2+),观察对针刺“内关”治疗心律失常效应的影响。结果表明,当络合掉“曲泽”穴处的 Ca(2+),观察对针刺“内关”治疗心律失常效应的影响。结果表明,当络合掉“曲泽”穴处的 Ca(2+)后,针刺“内关”治疗家兔实验性心律失常的效应不复存在。提示 Ca(2+)后,针刺“内关”治疗家兔实验性心律失常的效应不复存在。提示 Ca(2+)可能是针效产生的关键性因素之一,Ca(2+)可能是针效产生的关键性因素之一,Ca(2+)可能参与了经络活动。The experimental arrhythmic model was made with Aconitin in rabbits,The Ne-iguan(PC6)point was punctured to observe the influence on recovering time ofarrhythmia.After the curative effect of puncturing Neiguan(P6)was confirmed,Ca(2+)可能参与了经络活动。